Fish, heart and blood circulatory problems and mercury
Type of Spiritual Experience
There is something appallingly tragic about this observation.
A simple food like fish has the potential to heal heart disease, but as you will see from the other observations, our governments and our irresponsible dentists and industry magnates have allowed mercury to escape into the sea is such high levels that fish is gradually becoming as full of mercury as we are.
A description of the experience
J Clin Hypertens (Greenwich). 2011 Aug;13(8):621-7. doi: 10.1111/j.1751-7176.2011.00489.x. Epub 2011 Jul 11.
Role of mercury toxicity in hypertension, cardiovascular disease, and stroke.
- Department of Medicine, Vanderbilt University School of Medicine, Division of Human Nutrition, Saint Thomas Medical Group, Saint Thomas Hospital, Nashville, TN, USA. firstname.lastname@example.org
Mercury has a high affinity for sulfhydryl groups, inactivating numerous enzymatic reactions, amino acids, and sulfur-containing antioxidants (N-acetyl-L-cysteine, alpha-lipoic acid, L-glutathione), with subsequent decreased oxidant defense and increased oxidative stress.
Mercury binds to metallothionein and substitute for zinc, copper, and other trace metals, reducing the effectiveness of metalloenzymes.
Mercury induces mitochondrial dysfunction with reduction in adenosine triphosphate, depletion of glutathione, and increased lipid peroxidation.
Increased oxidative stress and reduced oxidative defense are common.
Selenium and fish containing omega-3 fatty acids antagonize mercury toxicity.
The overall vascular effects of mercury include increased oxidative stress and inflammation, reduced oxidative defense, thrombosis, vascular smooth muscle dysfunction, endothelial dysfunction, dyslipidemia, and immune and mitochondrial dysfunction.
The clinical consequences of mercury toxicity include hypertension, coronary heart disease, myocardial infarction, cardiac arrhythmias, reduced heart rate variability, increased carotid intima-media thickness and carotid artery obstruction, cerebrovascular accident, generalized atherosclerosis, and renal dysfunction, insufficiency, and proteinuria.
Pathological, biochemical, and functional medicine correlations are significant and logical.
Mercury diminishes the protective effect of fish and omega-3 fatty acids. Mercury inactivates catecholaminei-0-methyl transferase, which increases serum and urinary epinephrine, norepinephrine, and dopamine.
This effect will increase blood pressure and may be a clinical clue to mercury-induced heavy metal toxicity.
Mercury toxicity should be evaluated in any patient with hypertension, coronary heart disease, cerebral vascular disease, cerebrovascular accident, or other vascular disease. Specific testing for acute and chronic toxicity and total body burden using hair, toenail, urine, and serum should be performed.
© 2011 Wiley Periodicals, Inc.
The source of the experiencePubMed
Concepts, symbols and science items
Activities and commonsteps
Blood circulatory system disease
Heart failure and coronary heart disease
Heavy metal poisoning
Nervous system disease
Cod liver oil