Does heaven exist? With well over 100,000 plus recorded and described spiritual experiences collected over 15 years, to base the answer on, science can now categorically say yes. Furthermore, you can see the evidence for free on the website allaboutheaven.org.

Available on Amazon
also on all local Amazon sites, just change .com for the local version (.co.uk, .jp, .nl, .de, .fr etc.)


This book, which covers Visions and hallucinations, explains what causes them and summarises how many hallucinations have been caused by each event or activity. It also provides specific help with questions people have asked us, such as ‘Is my medication giving me hallucinations?’.

Available on Amazon
also on all local Amazon sites, just change .com for the local version (.co.uk, .jp, .nl, .de, .fr etc.)


Local anaesthetics

Category: Medicines


Involuntary and voluntary

Introduction and description

 see below for painting credits

Local anaesthetics are agents that prevent transmission of nerve impulses without causing unconsciousness. They act by binding to fast sodium channels from within (in an open state). Local anaesthetics can be either ester or amide based.

They are used to provide temporary local pain relief or to ensure absence of pain and nervous sensation in a specific location  during, for example, surgical procedures. Local anaesthetics can also cause paralysis – loss of muscle power – in selected areas.   For the purposes of pain relief, local anaesthetic drugs are often given by repeated injection or continuous infusion through a catheter. Low doses of local anaesthetic drugs can be sufficient so that muscle weakness does not occur.

Given the nature of the drugs, the likelihood of getting hallucinations is quite remote and indeed the evidence below bears this out, but there are some cases and as such they have been included so that the picture is complete.  Rather extraordinarily, they have also started to be a drug of abuse.

Some background


All local anaesthetics are ‘membrane stabilizing drugs’; they reversibly decrease the rate of depolarisation and repolarisation of excitable membranes. 

They act mainly by inhibiting sodium influx through sodium-specific ion channels in the neuronal cell membrane, in particular the so-called voltage-gated sodium channels. When the influx of sodium is interrupted, an action potential cannot arise and signal conduction is inhibited.

Local anaesthetics can block almost every nerve between the peripheral nerve endings and the central nervous system. The most peripheral technique is topical anaesthesia to the skin or other body surface. Small and large peripheral nerves can be anaesthetised individually (peripheral nerve block) or in anatomic nerve bundles (plexus anaesthesia). Spinal anaesthesia and epidurals merge into the central nervous system.

In June 2016, eHealthme ceased to provide the information on which all the data in this section is based.  On querying my friends in the USA, it would seem that many of the sites that provided similar information, have done the same.  The links we provided to eHealthme also no longer work as this data too has been removed. 

As to why all these sites have removed exceptionally important information, my USA helpers said that more and more people are questioning what they are being given – and demanding to know WHY the CAUSE of their illness has not been investigated.  It appears that there has been a very heartening increase in the numbers of people who want to be healed – have the cause tackled and not the symptoms.  And this is ‘not popular’ with the conventional medical community, who cannot make money from well people.

The statistics collected from eHealthme remain valid for the date they were collected.  As such we have left this section as it is – an historical record.  Please read this section therefore only as an historical record of the figures that were applicable on the date specified.

Side effects

Administered by properly trained competent medical staff, local anaesthetics generally do not seem to have extensive side-effects.  In some senses it is the correct use of a drug - a one off application to achieve healing.  Any drug that has to be taken every days is clearly not a healing drug by definition.   All we should expect from a pharmaceutical is that it has no deleterious side-effects, - it does no harm.  If we look at the detail below, the esters appear to be marginally better for not producing hallucinations than the amides. 


Lidocaine and its trade names

Lidocaine has a poor record of side effects and deaths, simply because it is sold over the counter.  The wisdom of doing this might be worth reconsidering.

Lidoderm is used to relieve post-shingles pain. Only apply Lidoderm to intact skin with no blisters.  Lidocaine topical in other formulations is used to reduce pain or discomfort caused by skin irritations such as sunburn, insect bites, poison ivy, poison oak, poison sumac, and minor cuts, scratches, hemorrhoids, and burns. Lidocaine topical can also be used to treat sores inside the mouth, during dental procedures to numb the gums, and to numb the skin for a medical procedure (such as getting stitches).
An overdose of Lidoderm can cause fatal side effects if too much lidocaine is absorbed through your skin and into your blood. This is more likely to occur when using a numbing medicine without the advice of a medical doctor (such as during a cosmetic procedure like laser hair removal). Overdose symptoms may include uneven heartbeats, seizure (convulsions), coma, slowed breathing, or respiratory failure (breathing stops). Your body may absorb more lidocaine if you use too much, if you apply it over large skin areas, or if you apply heat, bandages, or plastic wrap to treated skin areas. Skin that is cut or irritated may also absorb more topical medication than healthy skin.

 Lidoderm is self administration.  Some of the other deaths
may be attributable to its use as an antiarrhythmia
drug.  It is also being misused for GERD, plus a number
of other illnesses producing pain, all of which need to be
properly healed. Symptom suppression [as this is] is clearly
not healing and appears to be killing people.
  • On Sep, 18, 2015: 179 people reported to have side effects when taking Lidocaine. Among them, 5 people (2.79%) have Death
  • On Sep, 18, 2015: 1,107 people reported to have side effects when taking Lidocaine hydrochloride. Among them, 23 people (2.08%) have Death
  • On Sep, 18, 2015: 116 people reported to have side effects when taking Lidocaine hydrochloride in plastic container. Among them, 10 people (8.62%) have Death
  • On Sep, 18, 2015: 94 people reported to have side effects when taking Lidocaine viscous. Among them, 1 people (1.06%) has Death
  • On Sep, 16, 2015: 1,168 people reported to have side effects when taking Xylocaine. Among them, 12 people (1.03%) have Death
  • On Sep, 18, 2015: 170 people reported to have side effects when taking Xylocaine viscous. Among them, 2 people (1.18%) have Death
  • On Sep, 9, 2015: 3,196 people reported to have side effects when taking Lidoderm. Among them, 117 people (3.66%) have Death


Bupivacaine and trade names


The other drug in this list that appears to have a poor record of both hallucinations and deaths is Bupivacaine.  It belongs to the amide group. 

Compared to other local anaesthetics, bupivacaine is [and I quote] “markedly cardiotoxic.”   This same source states that “ adverse drug reactions (ADRs) are rare when it is administered correctly”.  But from what can be seen, it is not being administered correctly in some cases.

  • On Sep, 18, 2015: 1,419 people reported to have side effects when taking Sensorcaine. Among them, 8 people (0.56%) have Death.
  • On Sep, 2, 2015: 3,500 people reported to have side effects when taking Bupivacaine. Among them, 101 people (2.89%) have Death
  • On Sep, 18, 2015: 1,896 people reported to have side effects when taking Marcaine. Among them, 29 people (1.53%) have Death



Levobupivacaine belongs to the amino amide group and is marketed under the trade name Chirocaine.

Although the number of Adverse drug reactions (ADRs) is relatively speaking smaller, they too are linked to poor administration technique. 

Systemic exposure to excessive quantities of bupivacaine mainly result in central nervous system (CNS) and cardiovascular effects. CNS effects may include nervousness, tingling around the mouth, tinnitus, tremor, dizziness, blurred vision, seizures, drowsiness, loss of consciousness, respiratory depression and apnea. Cardiovascular effects include hypotension, bradycardia, arrhythmias, and/or cardiac arrest – some of which may be due to hypoxemia secondary to respiratory depression.

  • On Sep, 7, 2015: 107 people reported to have side effects when taking Chirocaine. Among them, 6 people (5.61%) have Death

Procaine and novocaine


Many of the hallucinations associated with procaine appear to be linked with its use in penicillin injections.  As this appears to be a very common problem, we have provided a number of observations to help in this area.  It has even been given the name Hoigne's syndrome.  Why we cannot fathom, as it simply masks the fact that this is an adverse  reaction between penicillin and novocaine that all doctors should be aware of:

  • On Sep, 18, 2015: 35 people reported to have side effects when taking Procaine hydrochloride. Among them, 1 people (2.86%) has Death
  • On Sep, 18, 2015: 128 people reported to have side effects when taking Novocain. Among them, 1 people (0.78%) has Death

Ropivacaine and Naropin


Almost exactly the same side effects occur with Naropin as they do with Chirocaine. It too belongs to the amino amide group.

  • On Sep, 18, 2015: 1,067 people reported to have side effects when taking Naropin. Among them, 20 people (1.87%) have Death








How it works

So, why the hallucinations?

Occasionally the muscle paralysis that works via local anaesthetics can affect the heart and the lungs and lead to - at worst  - a cardiac arrest and at best mild forms of hypoxia and as we saw in the section on hypoxia, this can cause a spiritual experience – often hallucinations.  The cardiac arrest has been known to produce near death experiences


Although local anaesthetics are intended to only affect the peripheral nervous system, depending on local tissue concentrations of the anaesthetic, there may be excitatory or depressant effects on the central nervous system. At lower concentrations, a relatively selective depression of inhibitory neurons results in cerebral excitation, which may lead to convulsions, again with possible effects spiritually – out of body experiences have been experienced.

A profound depression of brain functions at higher concentrations can lead to respiratory depression and hypoxia again – at too high a concentration it may lead to respiratory arrest and near death experiences. Such tissue concentrations may be due to very high plasma levels after intravenous injection of a large dose.  Ultimately this is an overdose.

In some cases the experience is due to an allergic reaction.  Adverse reactions to local anaesthetics (especially the esters) are not uncommon, but true allergy is very rare. Allergic reactions to the esters is usually due to a sensitivity to their metabolite, para-aminobenzoic acid (PABA), and does not result in cross-allergy to amides. There are also cases of allergy to paraben derivatives, which are - and I quote "often added as preservatives to local anesthetic solutions". 


Parabens are endocine disruptors, so they might be doing a great deal more harm than just giving us hallucinations.  Although the cause of any spiritual experience may vary, hypoxia is a common one.

One specific local anaesthetic has a reputation for inducing hypoxia. 

The metabolite of Prilocaine, o-toluidine, is known to cause methemoglobinemia, which reduces the amount of hemoglobin that is available for oxygen transport, ultimately this side effect is potentially life-threatening. 




References and further reading


The paintings on this page are by Leonor Fanny Borges Acevedo (1901 – 1998), better known by the pseudonym Norah Borges, sister of the  Argentinian writer Jorge Luis Borges. 

She was an artist, illustrator, graphic designer, set designer and art critic. 

Norah illustrated her brother’s book Cuaderno San Martin, for example, as she had done with his earlier works like Luna de enfrente , Fervor de Buenos Aires, and Las invitadas (1961).  She also illustrated  Autobiografia de Irene (1962) by Silvina Ocampo. Norah wrote as an art critic for Anales de Buenos Aires under the pseudonym Manuel Pinedo.

In 1942, a version of Platero y yo by Juan Ramon Jimenez was published with illustrations and vignettes by Norah.


She also worked as a graphic artist on other books by Spanish emigrants in Argentina like Ramón Gómez de la Serna, Rafael Alberti and León Felipe and illustrated the works of Argentinian writers like Victoria Ocampo, Adolfo Bioy Casares, Norah Lange and Julio Cortázar.

She worked as a journalist and painter until her death, but she gave away much of her work and did not care for regular art exhibitions. 

She died in Buenos Aires in 1998 and was buried in the family vault in the Cementerio de La Recoleta.

There is no reason for choosing her work to illustrate this page other than the fact we love her wistful, soulful paintings.


  • Manic reaction to lidocaine anesthesia.  Remick RA, Gimbarzevsky B.  J Clin Psychopharmacol. 1990 Dec;10(6):442-3. No abstract available.  PMID: 2286718


The following are examples of local anaesthetics, the figures show the known hallucinations that have occurred, derived  from the eHealthme web site. Each of the local anaesthetics have the suffix "-caine" in their names.  There are a large number with no record, but we have provided a link to eHealthme anyway so that you can see an up-to-date list of side effects.:


No of hallucinations

Articaine trade names Septocaine [with epinephrine], Zorcaine, Articadent [with epinephrine], Orabloc




Bupivacaine, Marcaine and Sensorcaine

18 + 10 = 28



Cinchocaine/Dibucaine brand names Cincain, Nupercainal, Nupercaine and Sovcaine



See separate entry







Lidocaine, Lignocaine, Xylocaine and Lidoderm

20 + 51 + 9 = 80

Levobupivacaine trade names Chirocaine


Mepivacaine marketed under various trade names including Carbocaine and Polocaine




Prilocaine and Citanest






Proparacaine and proxymetacaine


Ropivacaine and Naropin


Tetracaine/Amethocaine trade names Pontocaine. Ametop and Dicaine


Trimecaine  also Mesdicain, Mesocain, Mesokain and others




 The following is not intended to be a marketing statement but is there to indicate the chart above is missing some well used products because they are used outside the USA and th ehealthme figures are US based

In many anesthesia textbooks written in English, lidocaine, tetracaine, bupivacaine, ropivacaine, and chloroprocaine are listed as useful local anesthetics for spinal anesthesia. In contrast, T-cain is not included in these lists, even though it has been reported to be suitable for spinal anesthesia in Japan. T-cain was developed as a local anesthetic in the early 1940s by Teikoku Kagaku Sangyo Inc. in Itami, Japan, by replacing a methyl group on tetracaine (Pantocaine®) with an ethyl group. T-cain was clinically approved for topical use in Japan in November 1949, and a mixture of dibucaine and T-cain (Neo-Percamin S®) was approved for spinal use in May 1950. Simply because of a lack of foreign marketing strategy, T-cain has never attracted global attention as a local anesthetic. However, in Japan, T-cain has been used topically or intrathecally (as Neo-Percamin S®) for more than 60 years. Other than the side effects generally known for all local anesthetics, serious side effects have not been reported for T-cain. In fact, several articles have reported that T-cain decreases the neurotoxicity of dibucaine. In this historical review, the characteristics of T-cain and its rise to become a major spinal anesthetic in Japan are discussed.  PMID: 26302690



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