The knock on effects of mineral imbalance
Type of Spiritual Experience
An important background paper as to why hallucinations etc are caused by mineral imbalance.
As an additional example, increased extracellular potassium levels result in depolarization of the membrane potentials of cells. This depolarization opens some voltage-gated sodium channels, but not enough to generate an action potential. After a short while, the open sodium channels inactivate and become refractory, increasing the threshold needed to generate an action potential. This leads to the impairment of neuromuscular, cardiac, and gastrointestinal organ systems.
A description of the experience
Crit Care Resusc. 2002 Dec;4(4):307-15.
The essentials of calcium, magnesium and phosphate metabolism: part II. Disorders. Baker SB, Worthley LI; Department of Critical Care Medicine, Flinders University of South Australia, Adelaide, South Australia.
OBJECTIVE: To review the components of calcium, phosphate and magnesium metabolism that are relevant to the critically ill patient, in a two-part presentation.
DATA SOURCES: A review of articles reported on calcium, phosphate and magnesium disorders in the critically ill patient.
SUMMARY OF REVIEW: Abnormal calcium metabolism in the critically ill patient often presents with an alteration in plasma ionised calcium. The characteristic clinical features of an acute reduction in ionised plasma calcium include tetany, laryngospasm, paraesthesia, confusion, hallucinations, seizures and, rarely, hypotension ……………..
………. The clinical features of hypomagnesaemia include confusion, delerium, seizures, weakness, cramps, tetany and tachyarrhythmias, ……... …………
CONCLUSIONS: Calcium, phosphate and magnesium functions are closely linked with abnormal plasma levels of these compounds often causing similar cardiovascular and neurological features.